Core-periphery organization of complex networks

Networks may, or may not, be wired to have a core that is both itself densely connected and central in terms of graph distance. In this study we propose a coefficient to measure if the network has such a clear-cut core-periphery dichotomy. We measure this coefficient for a number of real-world and model networks and find that different classes of networks have their characteristic values. For example do geographical networks have a strong core-periphery structure, while the core-periphery structure of social networks (despite their positive degree-degree correlations) is rather weak. We proceed to study radial statistics of the core, i.e. properties of the n-neighborhoods of the core vertices for increasing n. We find that almost all networks have unexpectedly many edges within n-neighborhoods at a certain distance from the core suggesting an effective radius for non-trivial network processes

Virus Propagation in Multiple Profile Networks

Suppose we have a virus or one competing idea/product that propagates over a multiple profile (e.g., social) network. Can we predict what proportion of the network will actually get "infected" (e.g., spread the idea or buy the competing product), when the nodes of the network appear to have different sensitivity based on their profile? For example, if there are two profiles $\mathcal{A}$ and $\mathcal{B}$ in a network and the nodes of profile $\mathcal{A}$ and profile $\mathcal{B}$ are susceptible to a highly spreading virus with probabilities $\beta_{\mathcal{A}}$ and $\beta_{\mathcal{B}}$ respectively, what percentage of both profiles will actually get infected from the virus at the end? To reverse the question, what are the necessary conditions so that a predefined percentage of the network is infected? We assume that nodes of different profiles can infect one another and we prove that under realistic conditions, apart from the weak profile (great sensitivity), the stronger profile (low sensitivity) will get infected as well. First, we focus on cliques with the goal to provide exact theoretical results as well as to get some intuition as to how a virus affects such a multiple profile network. Then, we move to the theoretical analysis of arbitrary networks. We provide bounds on certain properties of the network based on the probabilities of infection of each node in it when it reaches the steady state. Finally, we provide extensive experimental results that verify our theoretical results and at the same time provide more insight on the problem

Edge overload breakdown in evolving networks

We investigate growing networks based on Barabasi and Albert's algorithm for generating scale-free networks, but with edges sensitive to overload breakdown. the load is defined through edge betweenness centrality. We focus on the situation where the average number of connections per vertex is, as the number of vertices, linearly increasing in time. After an initial stage of growth, the network undergoes avalanching breakdowns to a fragmented state from which it never recovers. This breakdown is much less violent if the growth is by random rather than preferential attachment (as defines the Barabasi and Albert model). We briefly discuss the case where the average number of connections per vertex is constant. In this case no breakdown avalanches occur. Implications to the growth of real-world communication networks are discussed.Comment: To appear in Phys. Rev.

Vertex overload breakdown in evolving networks

We study evolving networks based on the Barabasi-Albert scale-free network model with vertices sensitive to overload breakdown. The load of a vertex is defined as the betweenness centrality of the vertex. Two cases of load limitation are considered, corresponding to that the average number of connections per vertex is increasing with the network's size ("extrinsic communication activity"), or that it is constant ("intrinsic communication activity"). Avalanche-like breakdowns for both load limitations are observed. In order to avoid such avalanches we argue that the capacity of the vertices has to grow with the size of the system. An interesting irregular dynamics of the formation of the giant component (for the intrinsic communication activity case is also studied). Implications on the growth of the Internet is discussed.Comment: To appear in Phys. Rev.

Fast influence-based coarsening for large networks

Given a social network, can we quickly ‘zoom-out ’ of the graph? Is there a smaller equivalent representation of the graph that preserves its propagation characteristics? Can we group nodes together based on their influence properties? These are important problems with applications to influence analysis, epidemiology and viral marketing applications. In this paper, we first formulate a novel Graph Coarsening Problem to find a succinct representation of any graph while preserving key characteristics for diffusion processes on that graph. We then provide a fast and effective near-linear-time (in nodes and edges) algorithm coarseNet for the same. Using extensive experiments on multiple real datasets, we demonstrate the quality and scalability of coarseNet, en-abling us to reduce the graph by 90 % in some cases without much loss of information. Finally we also show how our method can help in diverse applications like influence maxi-mization and detecting patterns of propagation at the level of automatically created groups on real cascade data

Extinction Times of Epidemic Outbreaks in Networks

In the Susceptible–Infectious–Recovered (SIR) model of disease spreading, the time to extinction of the epidemics happens at an intermediate value of the per-contact transmission probability. Too contagious infections burn out fast in the population. Infections that are not contagious enough die out before they spread to a large fraction of people. We characterize how the maximal extinction time in SIR simulations on networks depend on the network structure. For example we find that the average distances in isolated components, weighted by the component size, is a good predictor of the maximal time to extinction. Furthermore, the transmission probability giving the longest outbreaks is larger than, but otherwise seemingly independent of, the epidemic threshold